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HPV modulation of host immune responses.
L. Feller
*
,
N. H. Wood
, R. A. Khammissa
, U. M. Chikte
, R. Meyerov
, J. Lemmer
*
Corresponding author for this work
Periodontology and Oral Medicine
Research output
:
Contribution to journal
›
Article
›
peer-review
16
Citations (Scopus)
Overview
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Keyphrases
HPV Infection
100%
Host Immune Response
100%
Keratinocytes
50%
Persistent Infection
50%
Suprabasal
50%
Vaccination
25%
Virus
25%
Immune Response
25%
Malignancy
25%
Age of Infection
25%
Viral Genome
25%
Genetic Makeup
25%
Squamous Cell Carcinoma (SqCC)
25%
Circulating Immune Cells
25%
Host Immune Evasion
25%
Specific T Cells
25%
Infected Cells
25%
HPV-related Cancers
25%
T Cell Immune Response
25%
Immune Surveillance
25%
High-risk HPV
25%
Active Immune Response
25%
Genomic Characteristics
25%
Cellular Immunity
25%
Initial Infection
25%
Neoplastic Lesions
25%
HPV DNA
25%
Premalignancy
25%
Post-mitotic
25%
Reinfection
25%
Non-haemolytic
25%
Host Immunity
25%
Viral Gene
25%
Basal Epithelial Cell
25%
Humoral Immune Response
25%
Viral Proteins
25%
Immunology and Microbiology
Immune Response
100%
Host Immune Response
100%
Keratinocyte
66%
T Cell
33%
Virus Gene
33%
Squamous Cell
33%
Immunocompetent Cell
33%
Humoral Immunity
33%
Viral Protein
33%
Immune Evasion
33%
Adoptive Immunity
33%
Immunosurveillance
33%